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Studies show that the XPC variants are associated with increased DNA adduct levels after aflatoxin B1 exposure, increasing liver cancer risk.[ref][ref][ref] This means that for people who both accumulate more DNA damage from mycotoxins (because of detox variants) and repair it less efficiently (because of XPC variants), the longterm cancer risk from chronic exposure is significantly higher

We tested human CRC cell models SW480 (a KRAS mutant, TP53 mutant MSS line

Mashayekhi M et al

These effects of macrophage produced NO explain, to a large degree, the cytotoxic and cytostatic effects of NO on invading parasites as well as on certain tumor cells